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*This document is like a lab demo—an example Certificate of Analysis (COA) that showcases what a COA looks like but may not match the latest batch in all its glory. Think of it as a chemistry experiment: close, but not quite the final reaction!
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Bulk Quote| precautionary statement codes | P203, P210, P233, P240, P241, P242, P243, P260, P261, P264, P264+P265, P270, P271, P280, P302+P352, P303+P361+P353, P304+P340, P305+P351+P338, P317, P318, P319, P321, P332+P317, P337+P317, P362+P364, P370+P378, P403+P235, P405, and P501 |
| hazard statements |
| hazards summary | Pure carbon disulfide is a colorless liquid with a pleasant odor that is like the smell of chloroform. The impure carbon disulfide that is usually used in most industrial processes is a yellowish liquid with an unpleasant odor, like that of rotting radishes. Carbon disulfide evaporates at room temperature, and the vapor is more than twice as heavy as air. It easily explodes in air and also catches fire very easily. In nature, small amounts of carbon disulfide are found in gases released to the earth’s surface as, for example, in volcanic eruptions or over marshes. Commercial carbon disulfide is made by combining carbon and sulfur at very high temperaturesExposure to carbon disulfide occurs mainly in the workplace. Acute (short-term) inhalation exposure of humans to carbon disulfide has caused changes in breathing and chest pains. Nausea, vomiting, dizziness, fatigue, headache, mood changes, lethargy, blurred vision, delirium, and convulsions have also been reported in humans acutely exposed by inhalation. Neurologic effects, including behavioral and neurophysiological changes, have been observed in chronic (long-term) human and animal inhalation studies. Reproductive effects, such as decreased sperm count and menstrual disturbances, have been observed in humans exposed to carbon disulfide by inhalation. Animal studies support these findings. EPA has not classified carbon disulfide for human carcinogenicity.Liquid causes first degree burns on short exposure. Carbon disulfide has good warning properties. its vapor is heavier than air. Elevation of liver function tests has been reported after acute exposure. It can cause both acute and chronic encephalopathy. Alice Hamilton described in the 1920s two cases, one of slowly developing paralysis of the legs, the other of rapidly developing manic-depressive insanity. There is limited evidence that CS2 can increase spontaneous abortion in humans and birth defects in animals. There is also evidence that CS2 can decrease sperm counts in experimental animals and reduce libido in exposed workers. The risk of death from coronary heart disease was increased 2.5 to 5 fold in a studies of viscose rayon workers. Viscous rayon workers suffered retinal hemorrhages and microvascular aneurysms. Optic nerve damage was also seen after heavy acute or chronic exposure. Vigliani studied over 380 cases in Italy that occurred in the decades prior to 1953. He concluded that chronic exposure to at least 45 ppm was necessary to cause intoxication. Heavy, chronic exposure to CS2 is associated with kidney disease as a secondary effect from the accelerated atherosclerosis. See No evidence of cardiovascular toxicity in workers exposed below 5 ppm carbon disulfide. |
